4l1d: Difference between revisions

<StructureSection load='4l1d' size='340' side='right'caption='[[4l1d]], [[Resolution|resolution]] 2.50&Aring;' scene=''>

<table><tr><td colspan='2'>[[4l1d]] is a 3 chain structure with sequence from [https://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=4L1D OCA]. For a <b>guided tour on the structure components</b> use [https://proteopedia.org/fgij/fg.htm?mol=4L1D FirstGlance]. <br>

</td></tr><tr id='resources'><td class="sblockLbl"><b>Resources:</b></td><td class="sblockDat"><span class='plainlinks'>[https://proteopedia.org/fgij/fg.htm?mol=4l1d FirstGlance], [http://oca.weizmann.ac.il/oca-bin/ocaids?id=4l1d OCA], [https://pdbe.org/4l1d PDBe], [https://www.rcsb.org/pdb/explore.do?structureId=4l1d RCSB], [https://www.ebi.ac.uk/pdbsum/4l1d PDBsum], [https://prosat.h-its.org/prosat/prosatexe?pdbcode=4l1d ProSAT]</span></td></tr>

[https://www.uniprot.org/uniprot/SCN3B_HUMAN SCN3B_HUMAN] Brugada syndrome. The disease is caused by mutations affecting the gene represented in this entry.

[https://www.uniprot.org/uniprot/SCN3B_HUMAN SCN3B_HUMAN] Modulates channel gating kinetics. Causes unique persistent sodium currents. Inactivates the sodium channel opening more slowly than the subunit beta-1. Its association with neurofascin may target the sodium channels to the nodes of Ranvier of developing axons and retain these channels at the nodes in mature myelinated axons (By similarity).

*[[Ion channels 3D structures|Ion channels 3D structures]]

[[Category: Homo sapiens]]

[[Category: Large Structures]]

[[Category: Chirgadze DY]]

[[Category: Jackson AP]]

[[Category: Klingauf J]]

[[Category: Namadurai S]]

[[Category: Rajappa R]]

[[Category: Stott K]]

[[Category: Weimhofer M]]