1o7d: Difference between revisions

No edit summary
No edit summary
Line 1: Line 1:
[[Image:1o7d.png|left|200px]]
{{STRUCTURE_1o7d|  PDB=1o7d  |  SCENE=  }}  
{{STRUCTURE_1o7d|  PDB=1o7d  |  SCENE=  }}  
===THE STRUCTURE OF THE BOVINE LYSOSOMAL A-MANNOSIDASE SUGGESTS A NOVEL MECHANISM FOR LOW PH ACTIVATION===
{{ABSTRACT_PUBMED_12634058}}


===THE STRUCTURE OF THE BOVINE LYSOSOMAL A-MANNOSIDASE SUGGESTS A NOVEL MECHANISM FOR LOW PH ACTIVATION===
==Disease==
[[http://www.uniprot.org/uniprot/MA2B1_BOVIN MA2B1_BOVIN]] Note=Defects in MAN2B1 are the cause of lysosomal alpha-mannosidosis (AM). AM is a lysosomal storage disease characterized by accumulation of unbranched oligosaccharide chains. The disease manifests itself by head tremor, aggressive tendency, ataxia, failure to thrive, and early death.


{{ABSTRACT_PUBMED_12634058}}
==Function==
[[http://www.uniprot.org/uniprot/MA2B1_BOVIN MA2B1_BOVIN]] Necessary for the catabolism of N-linked carbohydrates released during glycoprotein turnover.


==About this Structure==
==About this Structure==
Line 14: Line 16:


==Reference==
==Reference==
<ref group="xtra">PMID:012634058</ref><references group="xtra"/>
<ref group="xtra">PMID:012634058</ref><references group="xtra"/><references/>
[[Category: Alpha-mannosidase]]
[[Category: Alpha-mannosidase]]
[[Category: Bos taurus]]
[[Category: Bos taurus]]

Proteopedia Page Contributors and Editors (what is this?)Proteopedia Page Contributors and Editors (what is this?)

OCA