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==Structural and biochemical characterization of ClfB:ligand interactions== | ==Structural and biochemical characterization of ClfB:ligand interactions== | ||
<StructureSection load='3asw' size='340' side='right' caption='[[3asw]], [[Resolution|resolution]] 2.60Å' scene=''> | <StructureSection load='3asw' size='340' side='right'caption='[[3asw]], [[Resolution|resolution]] 2.60Å' scene=''> | ||
== Structural highlights == | == Structural highlights == | ||
<table><tr><td colspan='2'>[[3asw]] is a 2 chain structure with sequence from [ | <table><tr><td colspan='2'>[[3asw]] is a 2 chain structure with sequence from [https://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens] and [https://en.wikipedia.org/wiki/Staphylococcus_aureus_subsp._aureus_N315 Staphylococcus aureus subsp. aureus N315]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=3ASW OCA]. For a <b>guided tour on the structure components</b> use [https://proteopedia.org/fgij/fg.htm?mol=3ASW FirstGlance]. <br> | ||
</td></tr><tr id=' | </td></tr><tr id='method'><td class="sblockLbl"><b>[[Empirical_models|Method:]]</b></td><td class="sblockDat" id="methodDat">X-ray diffraction, [[Resolution|Resolution]] 2.6Å</td></tr> | ||
<tr id='resources'><td class="sblockLbl"><b>Resources:</b></td><td class="sblockDat"><span class='plainlinks'>[https://proteopedia.org/fgij/fg.htm?mol=3asw FirstGlance], [http://oca.weizmann.ac.il/oca-bin/ocaids?id=3asw OCA], [https://pdbe.org/3asw PDBe], [https://www.rcsb.org/pdb/explore.do?structureId=3asw RCSB], [https://www.ebi.ac.uk/pdbsum/3asw PDBsum], [https://prosat.h-its.org/prosat/prosatexe?pdbcode=3asw ProSAT]</span></td></tr> | |||
<tr id='resources'><td class="sblockLbl"><b>Resources:</b></td><td class="sblockDat"><span class='plainlinks'>[ | |||
</table> | </table> | ||
== Disease == | == Disease == | ||
[ | [https://www.uniprot.org/uniprot/K1C10_HUMAN K1C10_HUMAN] Congenital reticular ichthyosiform erythroderma;Annular epidermolytic ichthyosis;Epidermolytic ichthyosis. The disease is caused by mutations affecting the gene represented in this entry. The disease is caused by mutations affecting the gene represented in this entry. The disease is caused by mutations affecting the gene represented in this entry. | ||
== Function == | == Function == | ||
[ | [https://www.uniprot.org/uniprot/K1C10_HUMAN K1C10_HUMAN] | ||
==See Also== | ==See Also== | ||
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__TOC__ | __TOC__ | ||
</StructureSection> | </StructureSection> | ||
[[Category: | [[Category: Homo sapiens]] | ||
[[Category: | [[Category: Large Structures]] | ||
[[Category: | [[Category: Staphylococcus aureus subsp. aureus N315]] | ||
[[Category: | [[Category: Ganesh VK]] | ||
Latest revision as of 18:54, 4 October 2023
Structural and biochemical characterization of ClfB:ligand interactionsStructural and biochemical characterization of ClfB:ligand interactions
Structural highlights
DiseaseK1C10_HUMAN Congenital reticular ichthyosiform erythroderma;Annular epidermolytic ichthyosis;Epidermolytic ichthyosis. The disease is caused by mutations affecting the gene represented in this entry. The disease is caused by mutations affecting the gene represented in this entry. The disease is caused by mutations affecting the gene represented in this entry. FunctionSee Also |
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