Inflammation & Rheumatoid Arthritis: Difference between revisions

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Articles in Proteopedia concerning [[Inflammation & Rheumatoid Arthritis]] include:
Articles in Proteopedia concerning [[Inflammation & Rheumatoid Arthritis]] include:
* [[Aspirin effects on COX aka PGHS|Asprin Effects on Cyclooxygenase]]
* [[Aspirin effects on COX aka PGHS|Aspirin Effects on Cyclooxygenase]]
* [[Cyclooxygenase]]
* [[Cyclooxygenase]]
* [[Interleukin-10]]
* [[Interleukin-10]]
* [[Molecular_Playground/YKL-40|YKL-40]]
* [[Molecular_Playground/YKL-40|YKL-40]]
* [[Toll-like Receptors]]
* [[Toll-like Receptors]]
* [[Phospholipase A2]]
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Latest revision as of 15:40, 13 November 2019

Neutrophils Migrate from to Inflamed Tissue via Chemotaxis

Inflammation is part of the immune response by vascular tissue to harmful pathogens and cell damage. Upon infection or cell damage, numerous process take place such as vasodilation via the release of mediators such as bradykinins, and increased permeability of blood vessels to allow neutrophils to approach the site. Rheumatoid Arthritis, is a chronic inflammatory disorder that typically effects joints at early stages but can result in dangerous inflammation of the lungs and kidneys.


Articles in Proteopedia concerning Inflammation & Rheumatoid Arthritis include:


To view automatically seeded indices concerning Inflammation & Rheumatoid Arthritis See:


To View FDA Approved Treatments for Inflammation & Rheumatoid Arthritis See: Treatments

To view other Proteopedia pages about diseases & drug targets, See: Pharmaceutical Drug Targets

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David Canner, Wayne Decatur, Alexander Berchansky