Alpha-bungarotoxin: Difference between revisions
No edit summary |
No edit summary |
||
Line 27: | Line 27: | ||
==α7 nicotinic acetylcholine receptor binding== | ==α7 nicotinic acetylcholine receptor binding== | ||
Response to sensory stimuli and seizure genesis has been linked to nicotinic mechanisms<ref>PMID: 8478687</ref>, which are mediated by two major classes of receptors: Ganglionic type, and neuromuscular type, which pharmacological analysis of seizure genesis and habituation in the rat brain is thought to be mediated by the latter type <ref>PMID: 2813485</ref>. α-BGT demonstrates this relationship due to its prominent binding in the CA3 region of the hippocampus <ref>PMID: 626914</ref>. | Response to sensory stimuli and seizure genesis has been linked to nicotinic mechanisms<ref>PMID: 8478687</ref>, which are mediated by two major classes of receptors: Ganglionic type, and neuromuscular type, which pharmacological analysis of seizure genesis and habituation in the rat brain is thought to be mediated by the latter type <ref>PMID: 2813485</ref>. α-BGT demonstrates this relationship due to its prominent binding in the CA3 region of the hippocampus <ref>PMID: 626914</ref>. | ||
See also: | |||
*[[Receptor]] | |||
*[[Transmembrane (cell surface) receptors]] | |||
*[[Ionotropic receptors]] | |||
</StructureSection> | </StructureSection> | ||
==3D structures of Alpha-bungarotoxin== | ==3D structures of Alpha-bungarotoxin== |